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FREDA FRIEDMAN, PHD LCSW
PHD LCSW
Social Worker
NPI: 1235231846Individual
Specialties, Licenses & Credentials
Social WorkerPrimary
Social Worker
Code: 104100000X
IL149006392(IL)
Research & Publications (20)
The effect of sleep deprivation on fine motor coordination in obstetrics and gynecology residents.
PMID 18822404·Am J Obstet Gynecol·2008
8-other
Clinical implementation of prostate image guided radiation therapy: a prospective study to define the optimal field of interest and image registration technique using automated x-ray volumetric imaging software.
PMID 18473493·Technol Cancer Res Treat·2008
8-other
Site-specific phosphorylation of raf in cells containing oncogenic ras-p21 is likely mediated by jun-N-terminal kinase.
PMID 18316782·Ann Clin Lab Sci·2008
7-preclinical
Two peptides derived from ras-p21 induce either phenotypic reversion or tumor cell necrosis of ras-transformed human cancer cells.
PMID 18066549·Cancer Chemother Pharmacol·2008
8-other
The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 protein.
PMID 17569632·Front Biosci·2007
7-preclinical
Rapid conformational dynamics of cytochrome P450 2E1 in a natural biological membrane environment.
PMID 17176083·Biochemistry·2006
7-preclinical
PNC-28, a p53-derived peptide that is cytotoxic to cancer cells, blocks pancreatic cancer cell growth in vivo.
PMID 16688716·Int J Cancer·2006
7-preclinical
A public access defibrillation programme in non-inpatient hospital areas.
PMID 16563600·Resuscitation·2006
8-other
Functional interactions of Raf and MEK with Jun-N-terminal kinase (JNK) result in a positive feedback loop on the oncogenic Ras signaling pathway.
PMID 16086581·Biochemistry·2005
7-preclinical
An effector peptide from glutathione-S-transferase-pi strongly and selectively blocks mitotic signaling by oncogenic ras-p21.
PMID 15200055·Protein J·2004
7-preclinical
Loop domain peptides from the SOS ras-guanine nucleotide exchange protein, identified from molecular dynamics calculations, strongly inhibit ras signaling.
PMID 15200054·Protein J·2004
7-preclinical
Comparison of molecular dynamics averaged structures for complexes of normal and oncogenic ras-p21 with SOS nucleotide exchange protein, containing computed conformations for three crystallographically undefined domains, suggests a potential role of these domains in ras signaling.
PMID 15200053·Protein J·2004
7-preclinical
Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growth.
PMID 12783204·Cancer Chemother Pharmacol·2003
8-other
Preferential induction of necrosis in human breast cancer cells by a p53 peptide derived from the MDM2 binding site.
PMID 12629507·Oncogene·2003
7-preclinical
Identification of the site of inhibition of mitogenic signaling by oncogenic ras-p21 by a ras effector peptide.
PMID 12206511·J Protein Chem·2002
7-preclinical
Inhibition of ras-induced oocyte maturation by peptides from ras-p21 and GTPase activating protein (GAP) identified as being effector domains from molecular dynamics calculations.
PMID 12206510·J Protein Chem·2002
7-preclinical
Comparison of the average structures, from molecular dynamics, of complexes of GTPase activating protein (GAP) with oncogenic and wild-type ras-p21: identification of potential effector domains.
PMID 12206509·J Protein Chem·2002
8-other
Arginine to lysine 108 substitution in recombinant CYP1A2 abolishes methoxyresorufin metabolism in lymphoblastoid cells.
PMID 12023936·Br J Pharmacol·2002
4-observational
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 445 E OHIO ST, SUITE 250
CHICAGO, IL 60611 - Phone
- (312) 245-9501
Quick Facts
- NPI
- 1235231846
- Entity Type
- Individual
- Gender
- Female
- Medicare
- Not confirmed
- Specialties
- 1
- Locations
- 1
- Publications
- 20
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