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TADDESE DESTA, M.D.
M.D.
Gastroenterology Physician
NPI: 1346326246IndividualAccepts Medicare
Specialties, Licenses & Credentials
Specialist
Specialist
Code: 174400000X
A49164(CA)
Gastroenterology PhysicianPrimary
Internal Medicine — Gastroenterology
Code: 207RG0100X
A49164(CA)
CMS Specialties
PrimaryINTERNAL MEDICINE
Education
OREGON HEALTH SCIENCES UNIVERSITY SCHOOL OF MEDICINE
Class of 1989
Research & Publications (20)
FOXO1 plays an important role in enhanced microvascular cell apoptosis and microvascular cell loss in type 1 and type 2 diabetic rats.
PMID 19168598·Diabetes·2009
7-preclinical
Activation of the acquired immune response reduces coupled bone formation in response to a periodontal pathogen.
PMID 19050291·J Immunol·2008
7-preclinical
P. gingivalis and E. coli lipopolysaccharides exhibit different systemic but similar local induction of inflammatory markers.
PMID 18597607·J Periodontol·2008
4-observational
Diabetes-enhanced tumor necrosis factor-alpha production promotes apoptosis and the loss of retinal microvascular cells in type 1 and type 2 models of diabetic retinopathy.
PMID 18403591·Am J Pathol·2008
7-preclinical
Inhibition of experimental periodontitis by a topical boron-based antimicrobial.
PMID 18218841·J Dent Res·2008
4-observational
Fibroblast apoptosis induced by Porphyromonas gingivalis is stimulated by a gingipain and caspase-independent pathway that involves apoptosis-inducing factor.
PMID 17587332·Cell Microbiol·2007
8-other
Revealing the burden of maternal mortality: a probabilistic model for determining pregnancy-related causes of death from verbal autopsies.
PMID 17288607·Popul Health Metr·2007
8-other
Genome-wide atlas of gene expression in the adult mouse brain.
PMID 17151600·Nature·2007
7-preclinical
Immunization enhances inflammation and tissue destruction in response to Porphyromonas gingivalis.
PMID 16552059·Infect Immun·2006
7-preclinical
Diabetes enhances periodontal bone loss through enhanced resorption and diminished bone formation.
PMID 16723646·J Dent Res·2006
7-preclinical
Tumor necrosis factor-alpha mediates diabetes-enhanced apoptosis of matrix-producing cells and impairs diabetic healing.
PMID 16507891·Am J Pathol·2006
7-preclinical
Diabetes enhances mRNA levels of proapoptotic genes and caspase activity, which contribute to impaired healing.
PMID 16443785·Diabetes·2006
7-preclinical
Porphyromonas gingivalis fimbriae are pro-inflammatory but do not play a prominent role in the innate immune response to P. gingivalis.
PMID 15826373·J Endotoxin Res·2005
4-observational
Cytokine profiling of macrophages exposed to Porphyromonas gingivalis, its lipopolysaccharide, or its FimA protein.
PMID 15664935·Infect Immun·2005
7-preclinical
AMBAR as a public health index for assessment of malnutrition in children.
PMID 16895040·Ethiop Med J·2004
8-other
Diabetes prolongs the inflammatory response to a bacterial stimulus through cytokine dysregulation.
PMID 15191547·J Invest Dermatol·2004
7-preclinical
Diabetes alters the response to bacteria by enhancing fibroblast apoptosis.
PMID 15033911·Endocrinology·2004
7-preclinical
Diabetes causes decreased osteoclastogenesis, reduced bone formation, and enhanced apoptosis of osteoblastic cells in bacteria stimulated bone loss.
PMID 14525917·Endocrinology·2004
7-preclinical
Tumor necrosis factor modulates fibroblast apoptosis, PMN recruitment, and osteoclast formation in response to P. gingivalis infection.
PMID 11706944·J Dent Res·2001
7-preclinical
In vitro evaluation of antibiotic prophylaxis in the prevention of biliary stent blockage.
PMID 10699774·Gastrointest Endosc·2000
4-observational
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 292 EUCLID AVE, STE 115
SAN DIEGO, CA 92114 - Phone
- (619) 266-3332
Quick Facts
- NPI
- 1346326246
- Entity Type
- Individual
- Gender
- Male
- Medicare
- Accepted
- Specialties
- 2
- Locations
- 1
- Years in Practice
- 37
- Publications
- 20
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