Back to Search
PETER BUTTRICK, MD
MD
Cardiovascular Disease Physician
NPI: 1629182175IndividualAccepts Medicare
Specialties, Licenses & Credentials
Cardiovascular Disease PhysicianPrimary
Internal Medicine — Cardiovascular Disease
Code: 207RC0000X
45094(CO)
CMS Specialties
PrimaryCARDIOVASCULAR DISEASE (CARDIOLOGY)
Additional
CARDIAC ELECTROPHYSIOLOGYINTERNAL MEDICINE
Education
STATE UNIVERSITY OF NY UPSTATE MEDICAL UNIVERSITY
Class of 1979
Research & Publications (20)
Ablation of ventricular myosin regulatory light chain phosphorylation in mice causes cardiac dysfunction in situ and affects neighboring myofilament protein phosphorylation.
PMID 19106098·J Biol Chem·2009
7-preclinical
Maladaptation of calcium homoeostasis in aging cardiac myocytes.
PMID 18172603·Pflugers Arch·2008
4-observational
Principal strain changes precede ventricular wall thinning during transition to heart failure in a mouse model of dilated cardiomyopathy.
PMID 17965277·Am J Physiol Heart Circ Physiol·2008
7-preclinical
Recruitment of NADH shuttling in pressure-overloaded and hypertrophic rat hearts.
PMID 17229809·Am J Physiol Cell Physiol·2007
7-preclinical
PKCepsilon increases phosphorylation of the cardiac myosin binding protein C at serine 302 both in vitro and in vivo.
PMID 17503784·Biochemistry·2007
7-preclinical
Recruitment of compensatory pathways to sustain oxidative flux with reduced carnitine palmitoyltransferase I activity characterizes inefficiency in energy metabolism in hypertrophied hearts.
PMID 17404155·Circulation·2007
7-preclinical
The use of human cardiac tissue in biophysical research: the risks of translation.
PMID 17350034·J Mol Cell Cardiol·2007
6-review
The regulation of heat shock protein expression: how, when and where.
PMID 16982066·J Mol Cell Cardiol·2006
7-preclinical
The roles of corticotropin-releasing factor-related peptides and their receptors in the cardiovascular system.
PMID 16787262·Curr Protein Pept Sci·2006
6-review
Sex-related dimorphic response of HIF-1 alpha expression in myocardial ischemia.
PMID 16603692·Am J Physiol Heart Circ Physiol·2006
7-preclinical
Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylation.
PMID 16445938·J Mol Cell Cardiol·2006
4-observational
Depressed cardiac myofilament function in human diabetes mellitus.
PMID 16085678·Am J Physiol Heart Circ Physiol·2005
2-rct
Protein kinase C epsilon induces systolic cardiac failure marked by exhausted inotropic reserve and intact Frank-Starling mechanism.
PMID 15951344·Am J Physiol Heart Circ Physiol·2005
7-preclinical
Depressed cardiac tension cost in experimental diabetes is due to altered myosin heavy chain isoform expression.
PMID 15001437·Am J Physiol Heart Circ Physiol·2004
7-preclinical
Protein kinase Cepsilon overexpression alters myofilament properties and composition during the progression of heart failure.
PMID 15242976·Circ Res·2004
7-preclinical
Inhibition of PKC phosphorylation of cTnI improves cardiac performance in vivo.
PMID 14726296·Am J Physiol Heart Circ Physiol·2004
7-preclinical
Oxygen free radicals and heart failure: new insight into an old question.
PMID 12114183·Am J Physiol Lung Cell Mol Physiol·2002
6-review
Subtype specific roles of beta-adrenergic receptors in apoptosis of adult rat ventricular myocytes.
PMID 12099721·J Mol Cell Cardiol·2002
7-preclinical
alpha-Adrenergic response and myofilament activity in mouse hearts lacking PKC phosphorylation sites on cardiac TnI.
PMID 12003851·Am J Physiol Heart Circ Physiol·2002
7-preclinical
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 12605 E 16TH AVE
AURORA, CO 80045 - Phone
- (720) 848-0000
Quick Facts
- NPI
- 1629182175
- Entity Type
- Individual
- Gender
- Male
- Medicare
- Accepted
- Specialties
- 1
- Locations
- 1
- Years in Practice
- 47
- Publications
- 20
Are you this provider?
Claim Your Profile