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SCOTT MARSTON, M.D.
M.D.
Orthopaedic Surgery Physician
NPI: 1649244989IndividualAccepts Medicare
Specialties, Licenses & Credentials
Adult Reconstructive Orthopaedic Surgery Physician
Orthopaedic Surgery — Adult Reconstructive Orthopaedic Surgery
Code: 207XS0114X
42223(MN)
Orthopaedic Surgery PhysicianPrimary
Orthopaedic Surgery
Code: 207X00000X
42223(MN)
CMS Specialties
PrimaryORTHOPEDIC SURGERY
Education
UNIVERSITY OF MINNESOTA MEDICAL SCHOOL
Class of 1998
Research & Publications (20)
Genotype-phenotype correlations in ACTA1 mutations that cause congenital myopathies.
PMID 18976909·Neuromuscul Disord·2009
6-review
Evidence from human myectomy samples that MYBPC3 mutations cause hypertrophic cardiomyopathy through haploinsufficiency.
PMID 19574547·Circ Res·2009
8-other
Direct visualisation and kinetic analysis of normal and nemaline myopathy actin polymerisation using total internal reflection microscopy.
PMID 19418233·J Muscle Res Cell Motil·2009
7-preclinical
Back to the future: new techniques show that forgotten phosphorylation sites are present in contractile proteins of the heart whilst intensively studied sites appear to be absent.
PMID 19633912·J Muscle Res Cell Motil·2009
7-preclinical
Alpha-cardiac actin mutations produce atrial septal defects.
PMID 17947298·Hum Mol Genet·2008
7-preclinical
From genotype to phenotype: a longitudinal study of a patient with hypertrophic cardiomyopathy due to a mutation in the MYBPC3 gene.
PMID 19219553·J Muscle Res Cell Motil·2008
5-case
Myofilament dysfunction in cardiac disease from mice to men.
PMID 19140019·J Muscle Res Cell Motil·2008
6-review
How does genotype define phenotype? Microphysiology of a tropomyosin mutation in situ shows the limitations of reductionism.
PMID 18556722·J Physiol·2008
8-other
Myosin binding protein C phosphorylation in normal, hypertrophic and failing human heart muscle.
PMID 18573260·J Mol Cell Cardiol·2008
4-observational
The molecular phenotype of human cardiac myosin associated with hypertrophic obstructive cardiomyopathy.
PMID 18411228·Cardiovasc Res·2008
4-observational
Troponin phosphorylation and myofilament Ca2+-sensitivity in heart failure: increased or decreased?
PMID 18691597·J Mol Cell Cardiol·2008
7-preclinical
Role of caldesmon in the Ca2+ regulation of smooth muscle thin filaments: evidence for a cooperative switching mechanism.
PMID 17933868·J Biol Chem·2008
7-preclinical
Role of tropomyosin in the regulation of contraction in smooth muscle.
PMID 19209817·Adv Exp Med Biol·2008
6-review
The pathogenesis of ACTA1-related congenital fiber type disproportion.
PMID 17387733·Ann Neurol·2007
7-preclinical
Effect of mutations in the beta5-beta7 loop on the structure and properties of human small heat shock protein HSP22 (HspB8, H11).
PMID 17922839·FEBS J·2007
8-other
Troponin phosphorylation and regulatory function in human heart muscle: dephosphorylation of Ser23/24 on troponin I could account for the contractile defect in end-stage heart failure.
PMID 17081561·J Mol Cell Cardiol·2007
8-other
Small heat shock protein Hsp20 (HspB6) as a partner of 14-3-3gamma.
PMID 17109079·Mol Cell Biochem·2007
8-other
The effect of mutations in alpha-tropomyosin (E40K and E54K) that cause familial dilated cardiomyopathy on the regulatory mechanism of cardiac muscle thin filaments.
PMID 17360712·J Biol Chem·2007
7-preclinical
Fatal hypertrophic cardiomyopathy and nemaline myopathy associated with ACTA1 K336E mutation.
PMID 16945537·Neuromuscul Disord·2006
5-case
Structure and properties of K141E mutant of small heat shock protein HSP22 (HspB8, H11) that is expressed in human neuromuscular disorders.
PMID 16949546·Arch Biochem Biophys·2006
8-other
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 640 JACKSON ST
SAINT PAUL, MN 55101 - Phone
- (651) 254-2005
Quick Facts
- NPI
- 1649244989
- Entity Type
- Individual
- Gender
- Male
- Medicare
- Accepted
- Specialties
- 2
- Locations
- 1
- Years in Practice
- 28
- Publications
- 20
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