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JEFFREY TAVIS, M.D.
M.D.
Internal Medicine Physician
NPI: 1750568242Individual
Specialties, Licenses & Credentials
Internal Medicine PhysicianPrimary
Internal Medicine
Code: 207R00000X
A102252(CA)
Research & Publications (20)
Herpes simplex virus 2 UL13 protein kinase disrupts nuclear lamins.
PMID 19640559·Virology·2009
8-other
An interdomain RNA binding site on the hepadnaviral polymerase that is essential for reverse transcription.
PMID 19467554·Virology·2009
7-preclinical
Evidence for action of ribavirin through the hepatitis C virus RNA polymerase.
PMID 19243495·J Viral Hepat·2009
8-other
Loss of viral fitness and cross-recognition by CD8+ T cells limit HCV escape from a protective HLA-B27-restricted human immune response.
PMID 19139562·J Clin Invest·2009
8-other
Genome-wide hepatitis C virus amino acid covariance networks can predict response to antiviral therapy in humans.
PMID 19104147·J Clin Invest·2009
8-other
Pre-P is a secreted glycoprotein encoded as an N-terminal extension of the duck hepatitis B virus polymerase gene.
PMID 19004940·J Virol·2009
7-preclinical
Hepatitis C virus diversity and evolution in the full open-reading frame during antiviral therapy.
PMID 18463735·PLoS One·2008
8-other
Substrate specificity of the herpes simplex virus type 2 UL13 protein kinase.
PMID 18207213·Virology·2008
8-other
A novel unsupervised method to identify genes important in the anti-viral response: application to interferon/ribavirin in hepatitis C patients.
PMID 17589564·PLoS One·2007
8-other
Pretreatment sequence diversity differences in the full-length hepatitis C virus open reading frame correlate with early response to therapy.
PMID 17522222·J Virol·2007
3-trial
Changes in gene expression during pegylated interferon and ribavirin therapy of chronic hepatitis C virus distinguish responders from nonresponders to antiviral therapy.
PMID 17267482·J Virol·2007
8-other
Separation of near full-length hepatitis C virus quasispecies variants from a complex population.
PMID 17208310·J Virol Methods·2007
8-other
Combining genetic and biochemical approaches to identify functional molecular contact points.
PMID 17033698·Biol Proced Online·2006
8-other
The duck hepatitis B virus reverse transcriptase functions as a full-length monomer.
PMID 17005569·J Biol Chem·2006
7-preclinical
Suppression of mRNA accumulation by the duck hepatitis B virus reverse transcriptase.
PMID 16563457·Virology·2006
7-preclinical
A general method for nested RT-PCR amplification and sequencing the complete HCV genotype 1 open reading frame.
PMID 16321149·Virol J·2005
8-other
Identification of an essential molecular contact point on the duck hepatitis B virus reverse transcriptase.
PMID 16051809·J Virol·2005
8-other
Immune evasion versus recovery after acute hepatitis C virus infection from a shared source.
PMID 15939788·J Exp Med·2005
8-other
Herpes simplex virus 2 VP22 phosphorylation induced by cellular and viral kinases does not influence intracellular localization.
PMID 15527835·Virology·2004
7-preclinical
Detection and characterization of cytoplasmic hepatitis B virus reverse transcriptase.
PMID 15483251·J Gen Virol·2004
4-observational
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 12900 PARK PLAZA DR, STE 150
CERRITOS, CA 90703 - Phone
- (562) 741-4470
Quick Facts
- NPI
- 1750568242
- Entity Type
- Individual
- Gender
- Male
- Medicare
- Not confirmed
- Specialties
- 1
- Locations
- 1
- Publications
- 20
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