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VICKI ADLER, PH.D.
PH.D.
Psychologist
NPI: 1851564652Individual
Specialties, Licenses & Credentials
PsychologistPrimary
Psychologist
Code: 103T00000X
6301005484(MI)
Research & Publications (15)
The penetratin sequence in the anticancer PNC-28 peptide causes tumor cell necrosis rather than apoptosis of human pancreatic cancer cells.
PMID 18931881·Ann Surg Oncol·2008
8-other
Site-specific phosphorylation of raf in cells containing oncogenic ras-p21 is likely mediated by jun-N-terminal kinase.
PMID 18316782·Ann Clin Lab Sci·2008
7-preclinical
Two peptides derived from ras-p21 induce either phenotypic reversion or tumor cell necrosis of ras-transformed human cancer cells.
PMID 18066549·Cancer Chemother Pharmacol·2008
8-other
The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 protein.
PMID 17569632·Front Biosci·2007
7-preclinical
Alpha2-macroglobulin is a potential facilitator of prion protein transformation.
PMID 17453620·Amyloid·2007
7-preclinical
Two dual specificity kinases are preferentially induced by wild-type rather than by oncogenic RAS-P21 in Xenopus oocytes.
PMID 16720323·Front Biosci·2006
7-preclinical
Functional interactions of Raf and MEK with Jun-N-terminal kinase (JNK) result in a positive feedback loop on the oncogenic Ras signaling pathway.
PMID 16086581·Biochemistry·2005
7-preclinical
An effector peptide from glutathione-S-transferase-pi strongly and selectively blocks mitotic signaling by oncogenic ras-p21.
PMID 15200055·Protein J·2004
7-preclinical
Effector peptides from glutathione-S-transferase-pi affect the activation of jun by jun-N-terminal kinase.
PMID 15038666·Ann Clin Lab Sci·2004
7-preclinical
Small, highly structured RNAs participate in the conversion of human recombinant PrP(Sen) to PrP(Res) in vitro.
PMID 12946346·J Mol Biol·2003
7-preclinical
Concentration and removal of prion proteins from biological solutions.
PMID 12630906·Biotechnol Appl Biochem·2003
8-other
Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathway.
PMID 10854130·Cancer Chemother Pharmacol·2000
7-preclinical
p53 phosphorylation and association with murine double minute 2, c-Jun NH2-terminal kinase, p14ARF, and p300/CBP during the cell cycle and after exposure to ultraviolet irradiation.
PMID 10706102·Cancer Res·2000
8-other
Glutathione-S-Transferase as a selective inhibitor of oncogenic ras-p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinase.
PMID 10678584·Ann Clin Lab Sci·2000
7-preclinical
Analysis of JNK, Mdm2 and p14(ARF) contribution to the regulation of mutant p53 stability.
PMID 10656807·J Mol Biol·2000
8-other
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 74 W LONG LAKE RD, SUITE 104
BLOOMFIELD HILLS, MI 48304 - Phone
- (248) 642-6066
Quick Facts
- NPI
- 1851564652
- Entity Type
- Individual
- Gender
- Female
- Medicare
- Not confirmed
- Specialties
- 1
- Locations
- 1
- Publications
- 15
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