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LUCY CHIE, M.D.
M.D.
Obstetrics & Gynecology Physician
NPI: 1881709772IndividualAccepts Medicare
Specialties, Licenses & Credentials
Obstetrics & Gynecology PhysicianPrimary
Obstetrics & Gynecology
Code: 207V00000X
222251(MA)
CMS Specialties
PrimaryOBSTETRICS/GYNECOLOGY
Education
UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL
Class of 2000
Research & Publications (18)
Birth spacing and maternal risk of invasive epithelial ovarian cancer in a Swedish nationwide cohort.
PMID 18509730·Cancer Causes Control·2008
8-other
Cord serum estrogens, androgens, insulin-like growth factor-I, and insulin-like growth factor binding protein-3 in Chinese and U.S. Caucasian neonates.
PMID 18199728·Cancer Epidemiol Biomarkers Prev·2008
8-other
Specificity of inhibition of ras-p21 signal transduction by peptides from GTPase activating protein (GAP) and the son-of sevenless (SOS) ras-specific guanine nucleotide exchange protein.
PMID 16283548·Protein J·2005
7-preclinical
Functional interactions of Raf and MEK with Jun-N-terminal kinase (JNK) result in a positive feedback loop on the oncogenic Ras signaling pathway.
PMID 16086581·Biochemistry·2005
7-preclinical
An effector peptide from glutathione-S-transferase-pi strongly and selectively blocks mitotic signaling by oncogenic ras-p21.
PMID 15200055·Protein J·2004
7-preclinical
Loop domain peptides from the SOS ras-guanine nucleotide exchange protein, identified from molecular dynamics calculations, strongly inhibit ras signaling.
PMID 15200054·Protein J·2004
7-preclinical
Comparison of molecular dynamics averaged structures for complexes of normal and oncogenic ras-p21 with SOS nucleotide exchange protein, containing computed conformations for three crystallographically undefined domains, suggests a potential role of these domains in ras signaling.
PMID 15200053·Protein J·2004
7-preclinical
Oncogenic and activated wild-type ras-p21 proteins induce different isoforms of protein kinase C in mitogenic signal transduction.
PMID 14714729·J Protein Chem·2003
7-preclinical
A protein methyl transferase, PRMT5, selectively blocks oncogenic ras-p21 mitogenic signal transduction.
PMID 12817625·Ann Clin Lab Sci·2003
7-preclinical
Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growth.
PMID 12783204·Cancer Chemother Pharmacol·2003
8-other
Identification of the site of inhibition of mitogenic signaling by oncogenic ras-p21 by a ras effector peptide.
PMID 12206511·J Protein Chem·2002
7-preclinical
Inhibition of ras-induced oocyte maturation by peptides from ras-p21 and GTPase activating protein (GAP) identified as being effector domains from molecular dynamics calculations.
PMID 12206510·J Protein Chem·2002
7-preclinical
Comparison of the average structures, from molecular dynamics, of complexes of GTPase activating protein (GAP) with oncogenic and wild-type ras-p21: identification of potential effector domains.
PMID 12206509·J Protein Chem·2002
8-other
A protein kinase C inhibitor induces phenotypic reversion of ras-transformed pancreatic cancer cells and cooperatively blocks tumor cell proliferation with an anti- ras peptide.
PMID 12107546·Cancer Chemother Pharmacol·2002
4-observational
Differences in patterns of activation of MAP kinases induced by oncogenic ras-p21 and insulin in oocytes.
PMID 11525649·Exp Cell Res·2001
7-preclinical
Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathway.
PMID 10854130·Cancer Chemother Pharmacol·2000
7-preclinical
Glutathione-S-Transferase as a selective inhibitor of oncogenic ras-p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinase.
PMID 10678584·Ann Clin Lab Sci·2000
7-preclinical
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- SOUTH COVE COMMUNITY HEALTH CENTER, 885 WASHINGTON STREET
BOSTON, MA 02111 - Phone
- (617) 482-7555
Quick Facts
- NPI
- 1881709772
- Entity Type
- Individual
- Gender
- Female
- Medicare
- Accepted
- Specialties
- 1
- Locations
- 1
- Years in Practice
- 26
- Publications
- 18
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