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LEE KIEDROWSKI, MD
MD
Family Medicine Physician
NPI: 1962997338IndividualAccepts Medicare
Specialties, Licenses & Credentials
Family Medicine PhysicianPrimary
Family Medicine
Code: 207Q00000X
RL15208(ND)
CMS Specialties
PrimaryFAMILY PRACTICE
Education
UNIVERSITY OF NORTH DAKOTA SCHOOL OF MEDICINE
Class of 2018
Research & Publications (16)
AMPA-mediated excitotoxicity in oligodendrocytes: role for Na(+)-K(+)-Cl(-) co-transport and reversal of Na(+)/Ca(2+) exchanger.
PMID 17490438·J Neurochem·2007
7-preclinical
Critical role of sodium in cytosolic [Ca2+] elevations in cultured hippocampal CA1 neurons during anoxic depolarization.
PMID 17241128·J Neurochem·2007
7-preclinical
High activity of K+-dependent plasmalemmal Na+/Ca2+ exchangers in hippocampal CA1 neurons.
PMID 15486492·Neuroreport·2004
4-observational
Repolarization of the plasma membrane shapes NMDA-induced cytosolic [Ca2+ ] transients.
PMID 11733715·Neuroreport·2001
7-preclinical
Contribution of Na(+)/Ca(2+) exchange to excessive Ca(2+) loading in dendrites and somata of CA1 neurons in acute slice.
PMID 17598158·Hippocampus·2007
7-preclinical
Unconventional neuroprotection against Ca2+ -dependent insults by metalloporphyrin catalytic antioxidants.
PMID 16895586·J Neurochem·2006
7-preclinical
Importance of K+-dependent Na+/Ca2+-exchanger 2, NCKX2, in motor learning and memory.
PMID 16407245·J Biol Chem·2006
7-preclinical
Protection of ischemic rat spinal cord white matter: Dual action of KB-R7943 on Na+/Ca2+ exchange and L-type Ca2+ channels.
PMID 15755484·Neuropharmacology·2005
4-observational
Differential contribution of plasmalemmal Na/Ca exchange isoforms to sodium-dependent calcium influx and NMDA excitotoxicity in depolarized neurons.
PMID 15198672·J Neurochem·2004
4-observational
Inhibition of plasmalemmal Na(+)/Ca(2+) exchange by mitochondrial Na(+)/Ca(2+) exchange inhibitor 7-chloro-5-(2-chlorophenyl)-1,5-dihydro-4,1-benzothiazepin-2(3H)-one (CGP-37157) in cerebellar granule cells.
PMID 14637198·Biochem Pharmacol·2003
7-preclinical
Mitochondria buffer sodium-dependent CA2+ influx in cultured cerebellar granule cells.
PMID 12502589·Ann N Y Acad Sci·2002
7-preclinical
In depolarized and glucose-deprived neurons, Na+ influx reverses plasmalemmal K+-dependent and K+-independent Na+/Ca2+ exchangers and contributes to NMDA excitotoxicity.
PMID 12472886·J Neurochem·2002
7-preclinical
Preferential expression of plasmalemmal K-dependent Na+/Ca2+ exchangers in neurons versus astrocytes.
PMID 12218699·Neuroreport·2002
4-observational
Instrumental role of Na+ in NMDA excitotoxicity in glucose-deprived and depolarized cerebellar granule cells.
PMID 12064485·J Neurochem·2002
7-preclinical
Kainate-induced K+ efflux and plasma membrane depolarization in cultured cerebellar granule cells.
PMID 11201092·Neuroreport·2001
7-preclinical
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 701 E ROSSER AVE
BISMARCK, ND 58501 - Phone
- (701) 751-9500
Quick Facts
- NPI
- 1962997338
- Entity Type
- Individual
- Gender
- Male
- Medicare
- Accepted
- Specialties
- 1
- Locations
- 1
- Years in Practice
- 8
- Publications
- 16
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