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DR. JOSEPH NEALON SHAUGHNESSY M.D.
M.D.
Radiation Oncology Physician
NPI: 1134361371IndividualAccepts Medicare
Specialties, Licenses & Credentials
Radiation Oncology PhysicianPrimary
Radiology — Radiation Oncology
Code: 2085R0001X
01074457A(IN)46969(KY)35-129630(OH)
Education
UNIVERSITY OF LOUISVILLE SCHOOL OF MEDICINE
Class of 2009
Research & Publications (20)
A validated gene expression model of high-risk multiple myeloma is defined by deregulated expression of genes mapping to chromosome 1.
PMID 17105813·Blood·2007
8-other
Amplification and overexpression of CKS1B at chromosome band 1q21 is associated with reduced levels of p27Kip1 and an aggressive clinical course in multiple myeloma.
PMID 16188652·Hematology·2005
8-other
Interpreting the molecular biology and clinical behavior of multiple myeloma in the context of global gene expression profiling.
PMID 12846813·Immunol Rev·2003
6-review
Global gene expression profiling in the study of multiple myeloma.
PMID 12731663·Int J Hematol·2003
6-review
Prognostic impact of cytogenetic and interphase fluorescence in situ hybridization-defined chromosome 13 deletion in multiple myeloma: early results of total therapy II.
PMID 12492575·Br J Haematol·2003
8-other
The role of Dickkopf-1 in bone development, homeostasis, and disease.
PMID 18687985·Blood·2009
6-review
The role of IGF-1 as a major growth factor for myeloma cell lines and the prognostic relevance of the expression of its receptor.
PMID 19228610·Blood·2009
8-other
Inhibitor of DASH proteases affects expression of adhesion molecules in osteoclasts and reduces myeloma growth and bone disease.
PMID 19388929·Br J Haematol·2009
7-preclinical
High expression of BCL3 in human myeloma cells is associated with increased proliferation and inferior prognosis.
PMID 19191868·Eur J Haematol·2009
8-other
The ephrinB2/EphB4 axis is dysregulated in osteoprogenitors from myeloma patients and its activation affects myeloma bone disease and tumor growth.
PMID 19597185·Blood·2009
7-preclinical
Inhibiting Dickkopf-1 (Dkk1) removes suppression of bone formation and prevents the development of osteolytic bone disease in multiple myeloma.
PMID 19016584·J Bone Miner Res·2009
7-preclinical
Inhibition of aurora kinases for tailored risk-adapted treatment of multiple myeloma.
PMID 19171872·Blood·2009
8-other
Functional comparison of the binding of factor H short consensus repeat 6 (SCR 6) to factor H binding protein from Neisseria meningitidis and the binding of factor H SCR 18 to 20 to Neisseria gonorrhoeae porin.
PMID 19273554·Infect Immun·2009
8-other
Complete remission in multiple myeloma examined as time-dependent variable in terms of both onset and duration in Total Therapy protocols.
PMID 19515721·Blood·2009
3-trial
Cytogenetic abnormalities in multiple myeloma: poor prognosis linked to concomitant detection in random and focal lesion bone marrow samples and associated with high-risk gene expression profile.
PMID 19344415·Br J Haematol·2009
8-other
RARalpha2 expression is associated with disease progression and plays a crucial role in efficacy of ATRA treatment in myeloma.
PMID 19458357·Blood·2009
7-preclinical
Immunoglobulin isotypes in multiple myeloma: laboratory correlates and prognostic implications in total therapy protocols.
PMID 19120351·Br J Haematol·2009
8-other
Bortezomib induces osteoblast differentiation via Wnt-independent activation of beta-catenin/TCF signaling.
PMID 19196662·Blood·2009
7-preclinical
Gene expression profiles of tumor biology provide a novel approach to prognosis and may guide the selection of therapeutic targets in multiple myeloma.
PMID 19636021·J Clin Oncol·2009
8-other
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 3301 MERCY HEALTH BLVD STE 100
CINCINNATI, OH 45211 - Phone
- (513) 751-2273
Quick Facts
- NPI
- 1134361371
- Entity Type
- Individual
- Gender
- Male
- Medicare
- Accepted
- Specialties
- 3
- Locations
- 1
- Years in Practice
- 17
- Publications
- 20
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